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Thank You. This article is closed for comments. Please Kortak sound the Editor. This account emphasizes the role of top-down control processes mediated by higher brain functions and assumes deficits in descending auditory pathways. Empirical support for this proposal includes studies in humans that showed increased amplitudes of middle latency auditory evoked responses following lesions to the dorsolateral prefrontal cortex Knight kortak sound al. An alternative of this top-down account focuses on impaired inhibitory functions along the ascending auditory pathways as well as a loss of frequency selectivity.

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Evidence from animal studies has revealed a relationship between sensory-neural hearing loss and increased neural excitability within the ascending auditory pathway Willott and Lu, and primary auditory cortex Kotak et al. It is worth noting that we observed effects of mild hearing loss primarily on early exogenous evoked responses. This suggests that sensory-neural hearing loss impacts early cortical registration of acoustic information and further research is needed to determine whether even earlier stages of cortical processing would be impacted by hearing loss and how this relates to auditory perception and attention.

As in our previous aging kortak sound, we also found that the ORN amplitude increased with increasing mistuning.


Interestingly, the effect of kortak sound on perception was comparable in older adults with or without mild hearing loss. However, the ORN elicited by the mistuned harmonic was enhanced in older adults with mild hearing loss compared to normal hearing older adults. The effect of kortak sound hearing loss on both transient onset responses and ORN provides converging evidence for a general decline in inhibitory processes along the ascending auditory pathway, which likely contributes to increased time-locked cortical activity.


The peak latency of AEFs indicates the amount of time taken to generate the neuromagnetic response after sound onset and provides a means for assessing kortak sound of auditory processing. In the present study, the P1m latency was comparable for the two groups.

This suggests that conduction kortak sound in the ascending auditory pathways may be little affected by mild hearing loss. The N1m and P2m latencies were also comparable in older adults with and without mild hearing loss. Our findings are consistent with those of earlier Kortak sound studies using speech sounds Tremblay et al.

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